Tinnitaid

Tinnitaid

Calm the noise—anytime, anywhere.
A tinnitus relief companion that helps you match your tone and use sound-based relief on the go.

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How it works

Not medical advice. Tinnitaid is a wellness/relief tool and does not replace professional diagnosis or treatment.

tinnitus patient

Relief built for real life

Match your tone

Quickly detect and approximate the frequency you hear—then pick relief sounds aligned to it.

Relief anywhere

Use sound-based relief at work, in transit, or before sleep—no special equipment needed.

Doctor-led R&D

Built with ENT doctors, sound engineers, and researchers—grounded in therapy-inspired approaches.

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How it works

1) Detect

Use the tone tool to approximate the pitch you’re perceiving.

2) Choose

Select relief sounds tailored around your tone and preference.

3) Routine

Build a simple daily habit and track progress over time.

If you have sudden hearing loss, severe dizziness, or one-sided tinnitus, seek medical care promptly.

Awards / Investors / Partners

Kifissia Award

1st prize — Kifissia (Greece) entrepreneurship competition (2020)

Accelerace

Selected for the Accelerace startup accelerator

Are you an investor or clinical partner?

Email [email protected]

FAQ

Will this cure tinnitus?

Is it safe?

Who is it for?

Get support & updates

Questions, feedback, or want early access updates? Send a message—we reply quickly.

Latest articles

  • What Is Tinnitus?

    Tinnitus is the perception of sound in the absence of a corresponding external acoustic stimulus. With prevalence ranging from 10% to 15%, tinnitus is a common disorder. Many people habituate to the phantom sound, but tinnitus severely impairs quality of life of about 1-2% of all people. Tinnitus has traditionally been regarded as an otological disorder, but advances in neuroimaging methods and development of animal models have increasingly shifted the perspective towards its neuronal correlates. Increased neuronal firing rate, enhanced neuronal synchrony, and changes in the tonotopic organization are recorded in central auditory pathways in reaction to deprived auditory input and represent–together with changes in non-auditory brain areas–the neuronal correlate of tinnitus.

    References: Langguth B, Kreuzer PM, Kleinjung T, De Ridder D. Tinnitus: causes and clinical management. Lancet Neurol. 2013 Sep;12(9):920-930. doi: 10.1016/S1474-4422(13)70160-1. PMID: 23948178.

  • Is it tinnitus or is it hearing loss?

    Studies from independent labs demonstrated alterations in CN neural activity that were correlated with tinnitus behavior but not with changes in auditory brain stem response (ABR) thresholds or supra threshold ABR wave-1 amplitude (i.e., ABR responses to increasing levels of intensity) (Li et al., 2015; Wu et al., 2016).

    References: 1) Li S, Kalappa BI, Tzounopoulos T. Noise-induced plasticity of KCNQ2/3 and HCN channels underlies vulnerability and resilience to tinnitus. Elife. 2015 Aug 27;4:e07242. doi: 10.7554/eLife.07242. PMID: 26312501; PMCID: PMC4592936. 2) Wu C, Martel DT, Shore SE. Increased Synchrony and Bursting of Dorsal Cochlear Nucleus Fusiform Cells Correlate with Tinnitus. J Neurosci. 2016 Feb 10;36(6):2068-73. doi: 10.1523/JNEUROSCI.3960-15.2016. PMID: 26865628; PMCID: PMC4748084.