Tinnitaid

Tinnitaid

Calm the noise—anytime, anywhere.
A tinnitus relief companion that helps you match your tone and use sound-based relief on the go.

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How it works

Not medical advice. Tinnitaid is a wellness/relief tool and does not replace professional diagnosis or treatment.

tinnitus patient

Relief built for real life

Match your tone

Quickly detect and approximate the frequency you hear—then pick relief sounds aligned to it.

Relief anywhere

Use sound-based relief at work, in transit, or before sleep—no special equipment needed.

Doctor-led R&D

Built with ENT doctors, sound engineers, and researchers—grounded in therapy-inspired approaches.

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How it works

1) Detect

Use the tone tool to approximate the pitch you’re perceiving.

2) Choose

Select relief sounds tailored around your tone and preference.

3) Routine

Build a simple daily habit and track progress over time.

If you have sudden hearing loss, severe dizziness, or one-sided tinnitus, seek medical care promptly.

Awards / Investors / Partners

Kifissia Award

1st prize — Kifissia (Greece) entrepreneurship competition (2020)

Accelerace

Selected for the Accelerace startup accelerator

Are you an investor or clinical partner?

Email [email protected]

FAQ

Will this cure tinnitus?

Is it safe?

Who is it for?

Get support & updates

Questions, feedback, or want early access updates? Send a message—we reply quickly.

Latest articles

  • Is it tinnitus or is it hearing loss?

    Studies from independent labs demonstrated alterations in CN neural activity that were correlated with tinnitus behavior but not with changes in auditory brain stem response (ABR) thresholds or supra threshold ABR wave-1 amplitude (i.e., ABR responses to increasing levels of intensity) (Li et al., 2015; Wu et al., 2016).

    References: 1) Li S, Kalappa BI, Tzounopoulos T. Noise-induced plasticity of KCNQ2/3 and HCN channels underlies vulnerability and resilience to tinnitus. Elife. 2015 Aug 27;4:e07242. doi: 10.7554/eLife.07242. PMID: 26312501; PMCID: PMC4592936. 2) Wu C, Martel DT, Shore SE. Increased Synchrony and Bursting of Dorsal Cochlear Nucleus Fusiform Cells Correlate with Tinnitus. J Neurosci. 2016 Feb 10;36(6):2068-73. doi: 10.1523/JNEUROSCI.3960-15.2016. PMID: 26865628; PMCID: PMC4748084.

  • The Science Behind Tinnitus: What Happens Inside Your Ears

    Tinnitus results from changes within the auditory system, which may stem from damage to the ear or the neural pathways that carry sound to the brain. The most accepted theory is that tinnitus comes from outer hair cell damage in the cochlea in the ear, leading to changes in the signals sent from the ear to the brain. Some research suggests that when these normal signals are disrupted, the brain compensates by creating its own noise, leading to tinnitus. This phantom sound can also emerge as the brain’s auditory system becomes overly sensitive after hearing loss, detecting internal processes in the ear and interpreting them as sound. Further research indicates that tinnitus is associated with changes in the brain’s activity pattern, suggesting it’s not just a hearing issue but also involves significant neuroplastic changes within the brain’s auditory cortex (“Understanding the Facts,” American Tinnitus Association).

    Source: American Tinnitus Association – Understanding the Facts